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Transforming Growth Factor Beta Signalling in the Development of Muscle Weakness in Pulmonary Arterial Hypertension

24. september 2018 oppdatert av: Imperial College London
Pulmonary arterial hypertension (PAH) is a disease that causes raised blood pressure in blood vessels that pick up oxygen from the lungs. It has a life expectancy similar to some cancers. There is treatment available but there is no cure. We now know that PAH is associated with weakness in the muscles in the legs, which contributes to the symptoms patients' experience. Researchers believe that certain proteins found in high levels in the blood of patients with other chronic diseases can affect muscle function and growth. One of these proteins is called growth differentiating factor (GDF) 8, high levels of which are associated with muscle weakness in chronic obstructive pulmonary disease(COPD) and heart failure (HF). Interestingly there are drugs available which block the actions of GDF-8 on muscle cells which has been shown in animals to result in increased muscle size. A related protein called GDF-15 is found in elevated levels in patients PAH, and is linked to prognosis. Our preliminary data suggests that GDF-15 can also directly influence muscle size in a number of situations. We aim to investigate the role of GDF-15 and related molecules in the development of muscle weakness in patients with PAH. We will do this by measuring certain markers of muscle weakness and taking blood and muscle samples in patients and controls. We will then compare the levels of GDF-15 in these tissues in those with and without muscle wasting. We hope this work will lead to a greater understanding of the role of GDF-15 in the development of muscle weakness in patients with PAH. GDF-15 levels may be important in allowing us to define which patients have muscle weakness. In the future we aim to perform a clinical trial of drugs which block the actions of GDF-15.

Studieoversikt

Studietype

Observasjonsmessig

Registrering (Faktiske)

33

Kontakter og plasseringer

Denne delen inneholder kontaktinformasjon for de som utfører studien, og informasjon om hvor denne studien blir utført.

Studiesteder

      • London, Storbritannia, SW36NP
        • Royal Brompton Hospital

Deltakelseskriterier

Forskere ser etter personer som passer til en bestemt beskrivelse, kalt kvalifikasjonskriterier. Noen eksempler på disse kriteriene er en persons generelle helsetilstand eller tidligere behandlinger.

Kvalifikasjonskriterier

Alder som er kvalifisert for studier

14 år til 61 år (Voksen, Eldre voksen)

Tar imot friske frivillige

Nei

Kjønn som er kvalifisert for studier

Alle

Prøvetakingsmetode

Ikke-sannsynlighetsprøve

Studiepopulasjon

Clinics at Royal Brompton and Hammersmith hospital

Beskrivelse

Inclusion Criteria:

  • Patients with pulmonary arterial hypertension with New York Heart Association stage II - III disease will be eligible for recruitment in the patient portion of the trial. Interested healthy age matched volunteers will also be recruited.

Exclusion Criteria:

  • Patients and volunteers will be excluded if they have significant co-morbidities including other cardiorespiratory disease, metabolic abnormalities including diabetes or thyroid disorders. They will be excluded if they cannot safely exercise and perform a six minute walk test or if they are wheelchair bound.

Studieplan

Denne delen gir detaljer om studieplanen, inkludert hvordan studien er utformet og hva studien måler.

Hvordan er studiet utformet?

Designdetaljer

  • Observasjonsmodeller: Kohort
  • Tidsperspektiver: Annen

Kohorter og intervensjoner

Gruppe / Kohort
PH with wasting
This group of patients with idiopathic pulmonary arterial hypertension exhibit quadriceps wasting
PH no wasting
This groups of patients with idiopathic pulmonary arterial hypertension exhibit no evidence of muscle wasting
Controls
This group of volunteers does not have pulmonary arterial hypertension and would not be expected to have muscle wasting

Hva måler studien?

Primære resultatmål

Resultatmål
Tiltaksbeskrivelse
Tidsramme
Plasma growth and differentiation factor 15 levels in participants with and without muscle wasting
Tidsramme: 30 months
Muscle wasting will be defined by quadriceps cross sectional area measured by ultrasound
30 months

Sekundære resultatmål

Resultatmål
Tiltaksbeskrivelse
Tidsramme
Correlation of plasma Growth and differentiation factor 15 levels with muscle strength
Tidsramme: 30 months
Muscle strength will be measured by quadriceps maximal volitional capacity percent predicted
30 months
Change in fibre type in muscle biopsy
Tidsramme: 30 months
30 months
GDF-15 levels in biopsy specimens
Tidsramme: 30 months
30 months
Correlation of plasma Growth and differentiation factor 15 levels with brain natriuretic protein levels
Tidsramme: 30 months
30 months
Correlation of plasma Growth and differentiation factor 15 levels with fat free mass index
Tidsramme: 30 months
Fat free mass index will be measured by bioelectrical impedence
30 months
Correlation of plasma Growth and differentiation factor 15 levels with quality of life
Tidsramme: 30 months
Quality of life will be measured by St. George's respiratory questionnaire
30 months
Correlation of plasma Growth and differentiation factor 15 levels with exercise tolerance
Tidsramme: 30 months
Exercise tolerance will be measured by six minute walk test
30 months
Correlation of plasma Growth and differentiation factor levels 15 with physical activity levels
Tidsramme: 30 months
Physical activity will be measured by Sensewear armband
30 months
Correlation of plasma Growth and differentiation factor levels 15 with echocardiographic measures of severity of pulmonary hypertension
Tidsramme: 30 months
30 months
Correlation of GDF-15 levels in biopsy specimens with muscle wasting and weakness
Tidsramme: 30 months
Wasting will be measured by quadriceps cross sectional area and weakness will be defined by quadriceps maximal volitional capacity
30 months
Determine the contribution of atrophy and autophagy to muscle wasting in PAH
Tidsramme: 30 months
Muscle biopsy specimens will be evaluated using microscopy and real time polymerase chain reaction
30 months
Determine the contribution of SMAD and non-SMAD signalling pathways to the development of muscle weakness and wasting in PAH
Tidsramme: 30 months
Phosphorylation of SMAD and non-SMAD signalling will be determined by western blot
30 months

Samarbeidspartnere og etterforskere

Det er her du vil finne personer og organisasjoner som er involvert i denne studien.

Etterforskere

  • Hovedetterforsker: Stephen J Wort, MBBS, Imperial College / Royal Brompton Hospital

Publikasjoner og nyttige lenker

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Studierekorddatoer

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Studer hoveddatoer

Studiestart

1. oktober 2013

Primær fullføring (Faktiske)

1. august 2016

Studiet fullført (Faktiske)

1. august 2016

Datoer for studieregistrering

Først innsendt

19. april 2013

Først innsendt som oppfylte QC-kriteriene

2. mai 2013

Først lagt ut (Anslag)

7. mai 2013

Oppdateringer av studieposter

Sist oppdatering lagt ut (Faktiske)

26. september 2018

Siste oppdatering sendt inn som oppfylte QC-kriteriene

24. september 2018

Sist bekreftet

1. september 2018

Mer informasjon

Denne informasjonen ble hentet direkte fra nettstedet clinicaltrials.gov uten noen endringer. Hvis du har noen forespørsler om å endre, fjerne eller oppdatere studiedetaljene dine, vennligst kontakt register@clinicaltrials.gov. Så snart en endring er implementert på clinicaltrials.gov, vil denne også bli oppdatert automatisk på nettstedet vårt. .

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